Overexpression of miR-9-5p inhibits the expression level of GOT1 mRNA by direct binding to its 3’UTR, thus affecting the glutamine metabolism and redox homeostasis in pancreatic cancer cells, suggesting that miR-9-5p may serve as a prognostic or therapeutic target for pancreatic cancer [165]. This evidence concerns the gene GOT1 and familial pancreatic carcinoma.