Figures 4A–G shows that there was some effect on the high expression level of CDKN2A in seven tumors at several stages, specifically ACC, COAD, KICH, KIRC, KIRP, LIHC, and THCA. Moreover, CDKN2A with high expression levels in 10 tumors, BLCA, ACC, BRCA, THCA, PRAD, UCEC, LUAD, KICH, SKCM, KIRC, LIHC, and HNSC, was significantly correlated with TMB (Figure 4H), and high expression levels of CDKN2A in BRCA, UCEC and PRAD tumors were also significantly correlated with microsatellite instability (MSI) (Figure 4I). Here, CDKN2A is linked to prostate adenocarcinoma.