NFKB1 and endometritis: NF-κB is an essential regulator of the inflammatory response during endometritis [17].Our results indicated that the phosphorylatedNF-κB p65 (p-p65) level was obviously increased following S. aureus LTA stimulation (Figure 3(a)).Recent studies have reported that NF-κB is able to bind to the promoter region of miR-30 to induce its transactivation [27].