The previous study of our group showed that DOFP significantly increased the expression of occludin, claudin-1, and ZO-1 mRNA, tightened the loose arrangement of intestinal villi, protected the intestinal barrier function, and reduced the liver damage caused by lipopolysaccharide (LPS) in convalescent nonalcoholic fatty liver model mice (induced by a high-sugar and high-fat diet) [18]. The gene discussed is TJP1; the disease is non-alcoholic fatty liver.