This strong activation of IFN-γ following an exposition to M. tuberculosis and observations made on the antiviral immune response in pregnant woman seems to confirm the fact that pregnancy is not a period of immunosuppression notably for Th1 but a temporary attenuation in immune responses characterized by a strengthening of innate immune barriers and a concomitant reduction in pro-inflammatory immunity that is resumed when exposed to infections (21, 22). The gene discussed is IFNG; the disease is infection.