Moreover, there are no data on the temporal relationship between the composition and intensity of the TAB inflammatory infiltrate, duration of GCA and elevated levels of systemic inflammatory mediators and signs of inflammation in GCA patients, including erythrocyte sedimentation rate (ESR), C-reactive protein (CRP), acute phase serum amyloid A protein (A-SAA), peripheral blood thrombocytes, hemoglobin and fibrinogen (3, 5, 13). The gene discussed is SAA1; the disease is temporal arteritis.