STAT1 and neoplasm: A lot of these pro-tumoral genes overlapped with the group of potential tumor immune escape genes, of which 11 genes showed significantly increased gene expression under IFNγ treatment: CD74, STAT1, B2M, IRF1, WARS, IFI30, TAPBPL, IFIT1, PDIA3, ERAP1, and LGALS3BP.