The reason might be as follows: (a) MALT1 activated NF‐κB signaling pathway; meanwhile the activation of NF‐κB pathway was correlated with the secretion of various inflammatory cytokines (including IL‐1, IL‐6, TNF‐α, etc.)as well as the severity of intestinal inflammation.43, 44. This evidence concerns the gene NFKB1 and gastroenteritis.