Notably, amygdalin is nontoxic and has an anti‐liver fibrosis effect by decreasing the mRNA and protein expression levels of CTGF and TGF‐β (Luo et al., 2016), and inhibiting the expression of platelet‐derived growth factor and insulin‐like growth factor mRNA (Luo et al., 2018). Here, TGFB1 is linked to Hepatic fibrosis.