Given the simultaneous surge in both Th2 and IL-17 in the OVA + antigen group, IL-17 co-transferred with Th2 cytokines can promote eosinophilic airway inflammation mediated by Th2 cells by improving the expression of eotaxin and eotaxin-1 as major chemokines for recruitment of eosinophils into the airways [26–28]. The gene discussed is IL17A; the disease is inflammatory response.