While two studies found no significant difference in thrombosis recurrence among APS patients with and without thrombocytopenia [21, 22], a study of 138 patients with aPL positivity and thrombocytopenia (i.e., fulfilling laboratory but not clinical criteria of APS) described a five times higher risk of future thrombosis in these patients compared with those with normal platelet counts [23], and another publication described that, in aPL-positive patients, those with a low platelet count developed thrombosis more frequently than those without [24]. The gene discussed is FASLG; the disease is autoimmune polyendocrinopathy.