MTOR and gastric cancer: Xin Liu et al. showed that methyltransferase-like (METTL)-14 (an m6A writer) overexpression inhibited gastric cancer cell proliferation and invasion by regulating the phosphoinositide 3-kinase/serine-threonine kinase/mammalian target of rapamycin (PI3K/AKT/mTOR) signaling pathway [19].