To further ascertain whether Akt/mTOR signaling is associated with autophagy induced by sertaconazole, we transfected NSCLC cells with CA‐Akt (a constitutively active Akt) plasmids and found that sertaconazole‐induced LC3‐II levels and LC3 puncta were decreased after Akt reactivation in NSCLC cells (Figure S6C–E). The gene discussed is AKT1; the disease is non-small cell lung carcinoma.