Twelve KEGG pathways, “natural killer cell-mediated cytotoxicity,” “glycosaminoglycan biosynthesis chondroitin surface,” “antigen processing and presentation,” “regulation of autophagy,” “graft versus host disease,” “type I diabetes,” “T cell receptor signaling pathway,” “cytosolic DNA sensing pathway,” “TGF-β signaling pathway,” “chemokine signaling pathway,” “cytokine-to-cytokine receptor interaction,” and “toll-like receptor signaling pathway,” were enriched in the CCL4 high-expression BD patients (Figure 7). This evidence concerns the gene CCL4 and Behcet disease.