Additionally, elevated CCL4/MIP-1β in the cerebrospinal fluid is a characteristic of IgG4 anti‐neurofascin 155-positive chronic inflammatory demyelinating polyneuropathy, which implies exacerbation of protein leakage, damage of encephalic and CCL4/MIP-1β decreases with combined immune-treatment response (40). This evidence concerns the gene CCL4 and chronic inflammatory demyelinating polyradiculoneuropathy.