The number of mammalian achaete-scute homolog 1 (MASH1)-expressing type C cells was decreased by the anti-IL-10 antibody, while GFAP+/Nestin+ cells were increased at 4 days after stroke, suggesting that an IL-10-mediated neuroprotective effect may facilitate the proliferation of NSCs in the SVZ. Here, IL10 is linked to stroke disorder.