It causes alternative splicing of caspase 9 and Bcl-x with increasing pro-apoptotic Bcl-xS and caspase 9b mediated by the expression of protein phosphatase 1, leading to apoptosis in various human cancer cell lines (MCF7, A549, UACC903) (Sun Q. et al., 2018). This evidence concerns the gene BCL2L1 and cancer.