NR2F1 and Bosch-Boonstra-Schaaf optic atrophy syndrome: Early post-natal injection of Miconazole, a chemical drug promoting oligodendrocyte differentiation, efficiently rescued the hypomyelination phenotype of Nr2f1 haploinsufficient mice (Figure 7C; Bertacchi et al., 2019a), opening promising therapeutic avenues for BBSOAS visually-impaired patients and, more in general, supporting the feasibility of employing mouse models for therapeutic drug screening.