NFKB1 and Hepatic fibrosis: Additionally, we also studied the effect of uridine on NFκB signalling which is an important signalling pathway responsible regulating proinflammatory molecules expression, and it has been reported that NF‐κB signalling is closely related to liver fibrosis,27 and the results showed that CCl4 treatment activated NF‐κB, whereas uridine treatment inhibited the activation of NF‐κB (Figure 3C,D).