A cohort study that compared PET/CT findings in TB peritonitis and peritoneal carcinoma showed similar FDG uptake for the 2 entities, with no significant difference in SUV max.[23] TB lesions contain a large number of epithelioid cells, lymphocytes, and Langerhans cells that express high levels of glucose transporter 1 (Glut-1) and Glut-3, which induce high 18F-FDG uptake. This evidence concerns the gene SLC2A1 and tuberculosis.