We have previously shown that the pre-incubation of HPV16-PsVs with rhVim leads to a significant decrease in viral internalisation into the human keratinocyte cell lines HaCaT and NIKS as well as the human cervical cancer cell line HeLa, suggesting an inhibitory role of vimentin in the early events of the viral entry process [28]. Here, VIM is linked to cervical carcinoma.