In line, Gotthardt et al., highlighted the relationship between NK cells and angiogenesis, demonstrating that Stat5∆/∆Ncr1-iCreTg-Vav-Bcl2 mice failed to control tumor growth of RMA-S lymphoma cells and a v-abl transformed tumor, clarifying the significant tumor-promoting function of STAT5-deficient NK [48]. The gene discussed is ABL1; the disease is neoplasm.