This might signal during the early hours after infection stress (observed at 8 hpi by highly increased ASMTL and ADH5 transcription) relieve to the host cell (low oxidative stress indicated by ASMTL, SOD1 and SOD2), lower sugar concentration (less glycolysis combined with lower degree of fermentation observed from 24 hpi) and, thus, enable the normalization of TCA cycling and a decrease in energy depletion as it is SNRK-signaled after 24 hpi at 72 hpi before the expected burst of the classical immune system. The gene discussed is SOD1; the disease is infection.