However, the higher values for enolase and LDH observed at 24 hpi for cells infected by the mutant, the differential development of SNRK over time and higher values at 72 hpi observed for mTOR from donator 1 in the mutant (Figure 3) argue for prolonged cell cycle progression and a delay in cell cycle arrest related to both the origin of cells from donator 1 and infection by the mutant, respectively. The gene discussed is MTOR; the disease is infection.