CCKBR and neoplasm: In order to improve the tumor-specific uptake of [177Lu]Lu -PP-F11N, in a recent preclinical study, a kinase inhibitor library screen identified a clinically feasible way for augmented tumor uptake by the pharmacological inhibition of the mammalian target of rapamycin complex 1 (mTORC1), which enhanced the CCKBR protein level [8].