It has been reported that survivin enhances FasL expression in human colon cancer cells.[23] Others have found that, in esophageal squamous cell carcinoma, IL‐17A‐stimulated B cells gain more FasL expression,[24] which resembles our data on neutrophil's FasL regulation by Th17 cell‐derived IL‐17A in GC. Here, FASLG is linked to colonic neoplasm.