VEGFA and neoplasm: Li et al. (92) reported that cisplatin treatment induces upregulation of miR-29b, which suppressed invasion and angiogenesis of the cancer cells in vitro and inhibited tumor growth and neovascularization in vivo. The authors demonstrated that ectopic expression of miR-29b via intravenous administration in a subcutaneous xenograft mouse model of cervical cancer (HeLa cells) inhibited tumor growth and VEGF expression, corresponding to a decrease in vessel formation, although the authors did not evaluate this activity with the co-administration with cisplatin.