At present, the primary strategies for targeted tumor therapy include the following: (1) the expression of the proapoptotic gene p53 is restored [33]; (2) the antiapoptotic gene Bcl-2 is inactivated [34]; (3) the sensitivity of tumor cell apoptosis is increased by regulating metabolism (Bax and Bak) [35]; and (4) biological therapy is related (antibody-directed therapy, immunotherapy, virus-based introduction of apoptosis-inducing factor p53, and proapoptotic miRNA) [36–38]. This evidence concerns the gene BCL2 and neoplasm.