The ectopic expression of progerin, a mutant form of nuclear lamina protein A (LMNA) that causes accelerated aging in progeria, in an iPSC model of Parkinson's disease (PD) can trigger age‐related and degenerative phenotypes, including neuromelanin accumulation, dendrite degeneration, loss of tyrosine hydroxylase, and accumulation of pathological aggregates (Miller et al., 2013). This evidence concerns the gene TH and Parkinson disease.