MYOG and myotonic dystrophy type 1: Interestingly, KCa1.1 expression in myotonic dystrophy type 1 (DM1) myoblasts was found to be significantly decreased [95], whereas introducing functional KCa1.1 α-subunits into DM1 myoblasts reduced their proliferation to normal levels and rescued the expressions of MEF2 and myogenin [94].