CFTR and cystic fibrosis: Interestingly, upon infecting non-CF airway epithelial cells with Staphylococcus aureus or upon stimulating them with the proinflammatory cytokines TNF-α or IL-1β, the expression of these two miRNAs was increased, leading to a concurrent decrease in the CFTR expression and function, suggesting that inflammatory mediators may regulate these miRNAs.