In addition to the positive correlation between the PKCδ expression and STAT3 expression in patients with GBM (Figure 2C), the alleviation of PKCδ phosphorylation by magnolol implies that suppression of PKCδ phosphorylation may be linked to magnolol-inhibited STAT3 activation in GBM (Figure 3). The gene discussed is PRKCD; the disease is glioblastoma.