All pathways for TR2 vs. CTR were downregulated and immune associated (antigen processing and presentation; phagosome); the same was true for TR3 vs. CTR (antigen processing and presentation, graft-versus-host disease, allograft rejection, Epstein-Barr virus infection, and viral myocarditis). This evidence concerns the gene TXNRD2 and Epstein-Barr virus infection.