As reported by Hansen et al. in 2018, NO2-FAs can be formed in response to virus infection and are able to inhibit stimulator of interferon genes (STING) signaling, leading this way to an anti-inflammatory activity against several diseases, such as, systemic lupus erythematosus, Aicardi-Goutiéres syndrome and STING-associated vasculopathy [96]. The gene discussed is STING1; the disease is systemic lupus erythematosus.