Type of airway inflammation differs between asthmatic patients, but in childhood asthma up to 80% of patients demonstrates T helper cells subtype 2 (Th2)-mediated allergic inflammation with overexpression of Th2 cytokines (e.g., IL-4, IL-5 and IL-13), increased IgE production and enhanced recruitment of eosinophils to the site of inflammation in the airways [1]. Here, IGHE is linked to asthma.