CRP and myocardial infarction: During acute inflammatory cardiovascular events, such as thrombosis and myocardial infarction, the CRP levels increase rapidly, while activated platelets in blood vessels or cell necrosis caused by hypoxia in the heart provide abundant damaged membrane ligands for CRP dissociation, which leads to the accumulation of a large amount of ’active’ mCRP in the lesions within a short period of time [118,124].