IL17A and endothelial dysfunction: Since decreased Tregs and their specific cytokines such as IL-10 and TGFβ are impaired in pre-eclampsia, unrestricted T cells activation and differentiation into a pro-inflammatory phenotype support a pro-inflammatory environment with abundant TNFα, IL-6 and IL-17, advancing endothelial dysfunction, oxidative stress, B cells stimulation with high AT1-AA synthesis, and abnormal blood pressure.