Choi et al. [70] emphasized the key role of tumor-associated macrophages (TAMs) in BRCA, which also supports our results, which indicated that immune infiltration of M0 macrophages, M1 macrophages, and even M2 macrophages is highly correlated with SLC35A2 upregulation, and these TAMs have emerged as key players in tumor progression, with the potential as future treatment [71]. Here, SLC35A2 is linked to neoplasm.