In this survey of RTKs as regulatory controls of NAFLD, we arrive at a picture where activation of some RTKs (HGF, FGFR1) and inhibition of others (EGFR, AXL, FGFR4 and VEGFR) have found to be useful in tackling NAFLD. The gene discussed is KDR; the disease is metabolic dysfunction-associated steatotic liver disease.