In those diseases, the role of Pseudomonas aeruginosa favoring IgA secretion is not yet assessed and the potential role of other CF airway pathogens, such as Staphylococcus aureus, Haemophilus influenzae, and Burkholderia cepacia, in modulating S-IgA is not known and should be further studied. The gene discussed is CD79A; the disease is cystic fibrosis.