In addition, other disease-specific hiPSC-derived-CMs have also been produced for inherited arrhythmias, including various types of LQTS mutations, including, potassium channel inwardly rectifying channel subfamily J member 2 (KCNJ2) [208], calmodulin 1 (CALM1), or calmodulin 2 (CALM2) [209,210], sodium channel protein type 5 subunit alpha (SCN5A; Brugada syndrome type 1) [211,212]. This evidence concerns the gene SCN5A and familial long QT syndrome.