As expected, the hMXs + Dox (hMX1 + Dox and hMX3 + Dox) groups at 24 dpf developed palpable liver steatosis (Figure 4A, panels 4 and 6), whereas control groups (Figure 4A, panels 1–3 and 5) showed no or few fat accumulations. This evidence concerns the gene HMX1 and fatty liver disease.