BCL2 and myocardial infarction: Increases apoptosis, infarct size, and suppresses angiogenesis silencing sirtuin-1 and Wnt/β-catenin signaling pathway, suppresses cardiomyocyte proliferation and cardiac recovery post-MI regulating cell cycle activity and death via modulation of its targets, including Bcl2, Cyclin D1, and SIRT1 [90,91,184,185,186,187,188].