In addition, the second-mentioned study showed that AAB reactivities against the proteins 60 kDa heat shock protein (HSPD1/HSP60), previously identified by Wax et al., 1998 [109] in NTG patients, caldesmon (CALD1), voltage-dependent anion-selective channel protein 2 (VDAC2), and phosphoglycerate mutase 1 (PGAM1) were also increased in serum samples from glaucoma patients. The gene discussed is CALD1; the disease is glaucoma.