Taking these results together, on the one hand, quercetin could regulate the oxidative/anti-oxidative status to a more favorable physiological equilibrium in mice with psoriasis-like lesions, and, on the other hand, the inflammatory status of psoriatic mice was improved favorably after the treatment with quercetin, because of the capacity of quercetin to inhibit the activation of NF-κB through canonical and non-canonical NF-κB signaling [126]. This evidence concerns the gene NFKB1 and psoriasis.