RUNX1 and acute myeloid leukemia: Although KDM4B was not included as a component of major AE‐containing protein complexes according to the study, our results nevertheless show a certain genetic interaction between KDM4B and AML1‐ETO in promoting AML, and the gene expression analysis and ATAC‐seq in this study also support the functional involvement of KDM4B in transcriptional regulation mediated by AML‐ETO, suggesting that KDM4B presumably indirectly interacts with AE.