SEMA6B and neoplasm: As expected, we found that the SEMA6B high-risk group was markedly enriched in tumor cell proliferation, immune response, EMT-related pathways, such as the JAK-STAT signaling pathway, antigen processing and presentation, natural killer cell mediated cytotoxicity, the chemokine signaling pathway, cytokine-cytokine receptor interaction, ECM receptor interaction, and focal adhesion (Figure 7B).