CLHC1 and Aortic dissection: The efficiency of AAV-lnc-C2orf63-4-1 to rise endogenous lnc-C2orf63-4-1 in AAD mice was also confirmed by qRT-PCR, showing that the transcript level of lnc-C2orf63-4-1 in the aorta was significantly repressed in the aortic dissection group, while it was increased by AAV9-lnc-C2orf63-4-1 treatment (AAD + OElnc-V) (Figure 4A).