TNFRSF11B and neoplasm: In addition, six genes, KRT18, ARPC5L, ACTG1, ARPC2, EZR, and YWHAZ, were simultaneously enriched in tumors and tumor tissues highly expressing TNFRSF11B, which may enhance pathogenic E. coli focal adhesion, actin filament binding, and cadherin binding, as demonstrated by GO functional analysis.