Further potentially disease-relevant protein candidates without a known functional relation to hypertrophy, fibrosis or decompensated heart disease were SLTM, HNRNPLL, FIP1L1, RNMT, KRT18 and PUF60 and the downregulated NUDT4, GCAT, KIDINS220 and ARVCF. This evidence concerns the gene NUDT4 and heart disorder.