IL17A and psoriasis: A missense single‐nucleotide polymorphism (rs33980500; SNP‐D10N) in the coding region of ACT1 was suggested to increase the compensatory Th17 cells, with excessive secretion of IL‐22 and IL‐17 being the main cause of psoriasis susceptibility in ACT1‐D10N patients (Boisson et al, 2013).