Of the three CBPs defining GABAergic neurons in the human brain, parvalbumin (PV) is expressed relatively late during development after the establishment of synaptic contacts (Solbach and Celio 1991), leading us to hypothesise that there might be an early 'window of vulnerability' during which these neurons may be sensitive to e.g. excitotoxic damage, resulting in a deficit in these GABAergic cells in people with schizophrenia. Here, PVALB is linked to schizophrenia.